Subjective vertigo is when a person feels a false sensation of movement.
The neurochemistry of emesis overlaps with the neurochemistry of motion sickness and vertigo.
Vertigo is usually associated with a problem in the inner ear, or in the brain, or with the nerve connections between these two organs.
The most common cause of vertigo is benign paroxysmal positional vertigo or BPPV (Bellot and Mikhail 2005).
Vertigo-like symptoms may also appear as paraneoplastic syndrome (PNS) in the form of opsoclonus myoclonus syndrome, a multi-faceted neurological disorder associated with many forms of incipient cancer lesions or virus.
Vertigo is typically classified into one of two categories depending on the location of the damaged vestibular pathway.
Medical doctors (MDs) generally do not endorse this explanation of vertigo due to a lack of any data to support it, from an anatomical or physiological standpoint.
Serotonin is involved in central and peripheral control of emesis but has little influence on vertigo and motion sickness.
Objects immersed in liquids are subject to the phenomenon of buoyancy, which is also observed in other fluids, but is especially strong in liquids due to their high density.
Treatment is specific for underlying disorder of vertigo.
The onset of vertigo can be a symptom of an underlying harmless cause, such as cases of actual spinning, like the BPPV experienced from amusement rides.
Bleeding into the back of the brain (cerebellar hemorrhage) is characterized by vertigo, among other symptoms (Bellot and Mikhail 2005).